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[^1]: I would like to thank the editor for his/her assistance with this paper.

[^2]: D.N. Bernstein, personal communication.
Myocardial-nerve-evoked contractions in isolated guinea-pig ventricular strips.
The role of vagal innervation in the ventricular myocardium is not fully understood. It is known that in isolated cardiac strips at low stimulation frequencies (less than 2 Hz) they do not contract. However, when heart-nerve (HN) contractions were elicited at low frequencies by electrical stimulation of the HN, it was observed that a large proportion of the strips (approximately 20-50%) did not contract, regardless of whether the HN were perfused extracardiacly, or were entirely contained within the chest. Experiments in which the HN, identified by lucidiagnostic dye, were completely contained within the chest, but electrically stimulated externally to the heart, showed that at low HN stimulation frequencies, the HN contracted the heart. A similar response was recorded when the HN were stimulated directly to the heart from the extracardiac surface. It is suggested that vagal innervation may play a role in determining whether the myocardial strips contract in a given preparation or not.-β or MMP-2-deficient mice. They observed that the rate of graft vascularization was increased in both RA-NOG and MMP-9-KO-NOG mice as compared with NOG mice \[[@B98-ijms-21-00448]\]. The mechanism described was a reduction in intragraft accumulation of MMP-9-deficient CD4^+^ T cells. These data indicated that MMPs, particularly MMP-9, could influence graft vascularization and immune cell infiltration after transplantation.

Sherry et al. \[[@B99-ijms-21-00448]\] also studied the effects of MMP-2 and MMP-9 on the rate of renal graft vascularization in a mouse renal transplant model

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